Gall bladder disorders

Gallstones

Epidemiology

Present in 10 – 20% of the population in the West

More common in young women than young men, difference decreases with age

Rare in Africa

 

Types of gallstone

Composed of cholesterol and bile pigments – 80% 0f stones

Pure cholesterol stones are often solitary and less common

Bile pigment and calcium stone occur in people with increased haemolysis

 

Pathophysiology

Cholesterol based stones form when there is an excess of cholesterol over bile salts in the bile

Cholesterol crystals form and acts as an attraction for more precipitation

 

Aetiology

Essentially unknown, probably related to metabolic factors

 

Risk factors

Age and sex                                      Multiparity                               Obesity

High animal fat diet                           Diabetes                                Oral contraception-

 

Clinical picture

80% of gallstones remain in the gall bladder and are asymptomatic

18% have symptoms over a 15 year period

Stone may impact in the neck of the gallbladder or in the cystic bile duct ---------- biliary pain, Cholecystitis

Stasis may lead to bacterial infection ------- cholangitis

Pain in epigastrium and right hypocondrium

 

Stone in the common bile duct

Biliary obstruction  --------- severe biliary pain, cholestatic jaundice

Classical triad - abdominal pain, jaundice, fever

Vomiting

Pain for a few hours, returning after days, weeks or months

Leucocytosis                                                 Blood culture may grow gut organisms

Ultrasound                                                      Endoscopic retrograde cholangiopancreatography

 

Complications of gallstones

Pancreatitis               Fistula formation       Gallstone ileus           Carcinoma of the gall bladder

 

Management

Cholecystectomy - laproscopic if possible

Some cholesterol only stones may be dissolved with medication

Lithotripsy

Stones in the CBD can be removed by endoscopic sphincterotomy, if necessary followed by removal, or surgically

 

Inflammatory conditions

Acute cholecystitis

 

Pathophysiology

90% of cases caused by gallstone obstruction in neck or cystic areas ------------ distension and inflammation

Usually infected with gut organisms within 24 hours

Condition varies but is usually severe

Gall bladder wall involvement -------- localised peritonitis and acute pain

Empyema may occur

Acute gangrenous cholecystitis will lead to generalised peritonitis

 

Clinical features

Pain may radiate through to back and shoulder               

Severe continuous pain increasing in intensity over 24 hours

Possible nausea vomiting

Possible mild jaundice from inflammatory obstruction

Fever                          Shallow respirations             Guarding and rebound         Leucocytosis

Features seen on ultrasound

 

Management

Rest                            NBM                           IVIs                              Antibiotics                  Analgesia

Oral fluids when tolerated                             Cholecystectomy after a few days

 

Acute cholangitis

Bile duct abnormality leads to bacterial infection

Ductal pathology includes stones, strictures

Fever, upper abdominal pain, jaundice

Possible septicaemia

Treat with iv antibiotics

 

Primary sclerosing cholangitis

Inflammation and sclerosing of the bile ducts

Might be immunological                                           No specific treatment

 

Malignant conditions

 

Primary carcinoma of the gall bladder

1 % of cancers                                                                      Mostly over 70 years of age

More common in females                                                    Possible relationship with gallstones

Jaundice, pain, possible palpable mass

Cancers of the bile ducts or ampula occasionally present