Pharmacology and continence

 

This topic will be addressed in terms of drugs which may be used to treat incontinence and those which may be aetiological. 

 

Treatments

 

Laxatives

 

Bulk laxatives, (lumen acting)  

 

*           Increase volumes in the gut so stimulate peristalsis

*           Indigestible polysaccharides, methylcellulose and lactulose.

*           Osmotic agents, magnesium sulphate, epsom salts

 

Stimulant laxatives, (wall acting)

 

*           Increase motility, often acting on nerve plexuses

*           May cause abdominal cramps

*           Bisacodyl, senna, cascara

 

Lubricants, (mucosa acting)

 

*           Soften and/or lubricate

*           Dioctyl sodium, sulphosuccinate, liquid paraffin

 

 

Antidiarrhoeal drugs

 

Rehydration therapy

 

*           Water glucose and electrolytes

 

Antimotility drugs

 

*           Morphine, dipheroxylate and codeine inhibit ACh release from the       myenteric plexus

*           Loperamide is best for GI action as it does not easily enter the brain

 

Absorbants

 

*           Kaolin, little evidence of effectiveness

 

Antibiotics

 

*           Eg. tetracycline for severe bacterial dysentry and cholera. 

*           Most diarrhoea is viral in the UK, but remember protozoa

 

Antimicrobial drugs

 

Factors

 

*           Sensitivity

 

*           Resistance

 

*           Compliance

 

Inhibitors of nucleic acid synthesis

 

*           Sulphonamides, trimethoprim, quinolones

 

Inhibitors of cell wall synthesis

 

*           Penicillins, cephlosporins and vancomycin

*           Peptidoglycan transpeptidase

 

Inhibitors of bacterial protein synthesis

 

*           Aminoglycosides, tetracyclines, chloramphenicol, erythromycin

 

 

Anticholinergic and antispasmodics

 

Indicated in frequency, urgency and incontinence

 

Lower intravesicular pressure so increase capacity

 

Reduce frequency of contraction by blocking parasympathetic innervation

 

Some have a direct spasmolytic effect on the detrusor muscle.

 

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Cholinergics and anticholinesterases

 

Used in urinary retention

 

Promote voiding by increasing detrusor tone

 

Stimulate cholinergic receptors in other organs

 

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Selective alpha 1 blockers

 

Symptomatic treatment of benign prostatic hypertrophy, (BPH)

 

In BPH increased sympathetic tone constricts muscle in the urethra and prostate gland

 

These drugs remove this component of the obstruction

 

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5 - alpha reductase inhibitor

 

Indicated in BHP

 

Blocks the formation of dihydrotestosterone, therefore induces shrinkage of the hyperplastic prostatic tissue

 

 

Vasopressin analogue

 

In enuretic patients levels of ADH have been shown to be low

 

Supplementation of the natural hormone prevents overnight polyuria

 

May also be of benefit in multiple sclerosis

 

No effects occur on vasotone or on smooth muscle

 

 

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Antifungal and antibacterial

 

Preparations used for bladder instillation or irrigation

 

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Local anaesthetics

 

Concentration

Route

Time to take effect

 

Aetiological factors

 

Diuretics

 

Thiazides

*           Act on distal tubule to inhibit NaCl reabsorption

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Loop Diuretics

 

*           Act on thick ascending loop and distal tubule to inhibit NaCl reabsorption

 

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Potassium sparing diuretics

 

*           Act on aldosterone responsive segments of distal nephron where K+   homeostasis is controlled

 

*           Spirolactone, amiloride, triamtereme, alcohol

 

 

Anticholinergic

 

Reduce the cholinergically mediated parasympathetic supply to the gut. Possibility of GI obstruction in extreme cases.

 

Other possible drugs which may effect continence

 

Sedatives,  Beta blockers, Analgesics, Anti-diabetic, Anti-Parkinsons,

Oestrogen replacement